- Détails
- Catégorie : Actualités
- mardi 21 février 2017 14:13
Call for teams_en
We are looking for scientists with strong potential who are willing to lead an independent team in the sensory-motor and related fields. Young scientists will be encouraged to apply for permanent position in French research institutions (Inserm, Cnrs) as Avenir-Atip teams.
The Institute for Neurosciences of Montpellier (INM) comprises a staff of 200 personnel working on sensory and motor deficits. Research is devoted to gene discovery and function, development, neurotransmission and molecular mechanisms of sensory motor disorders. The INM houses teams working on vision, hearing, somatosensory system, myelination, motoneuron disorders, neural circuits and plasticity. Team competences cover expertise in genetic, molecular and cellular biology, electrophysiology, computational neurosciences and pre-clinical and clinical researches
The Institute harbors numerous technical platforms to carry-out molecular to systems level investigations, including fast confocal and multiphoton multicolor microscope for live animal imaging, correlative 3D-electron microscopy and sensory-motor behavioral analysis. Translational research is supported through many interactions between the INM and the clinical departments of the nearby University Neuroscience hospitals.
Contact
Please send your applications (CV and one page of research project) to:
jean-luc.puel @inserm.fr
- Détails
- Catégorie : Actualités
- dimanche 13 janvier 2013 18:50
Relevant Publications (2009-2015)
Bouilloux et al., Loss of the transcription factor Meis1 prevents sympathetic neurons target-field innervation and increases susceptibility to sudden cardiac death. Elife. 2016 Feb 8;5. pii: e11627.
Guillet et al., Actin Filaments Regulate Exocytosis at the Hair Cell Ribbon Synapse. J Neurosci. 2016 Jan 20;36(3):649-54.
Herbet et al., The dorsal cingulate cortex as a critical gateway in the network supporting conscious awareness. Brain. 2015 Dec 30. pii: awv381.
Angebault et al., Recessive. Mutations in RTN4IP1 Cause Isolated and Syndromic Optic Neuropathies. Am J Hum Genet. 2015 Nov 5;97(5):754-60.
Bowerman et al., Tweak regulates astrogliosis, microgliosis and skeletal muscle atrophy in a mouse model of amyotrophic lateral sclerosis. Hum Mol Genet. 2015 Jun 15;24(12):3440-56.
Ohayon et al., Zeb family members and boundary cap cells underlie developmental plasticity of sensory nociceptive neurons. Dev Cell. 2015 May 4;33(3):343-50.
Parsadaniantz et al., Opioid and chemokine receptor crosstalk: a promising target for pain therapy? Nat Rev Neurosci. 2015 Feb;16(2):69-78.
Guichet et al., Notch1 stimulation induces a vascularization switch with pericyte-like cell differentiation of glioblastoma stem cells. Stem Cells 2015 Jan;33(1):21-34.
Meunier et al., Frequency and clinical pattern of vitelliform macular dystrophy caused by mutations of interphotoreceptor matrix IMPG1 and IMPG2 genes. Ophthalmology 2014 Dec;121(12):2406-14.
Otsmane et al., Somatic and axonal LIGHT signaling elicit degenerative and regenerative responses in motoneurons, respectively. EMBO Rep. 2014, 15(5):540-7.
Sendin et al., Spatiotemporal pattern of action potential firing in developing inner hair cells of the mouse cochlea. Proc Natl Acad Sci U S A. 2014, 111(5):1999-2004.
Gonzalez et al., In vivo introduction of transgenes into mouse sciatic nerve cells in situ using viral vectors. Nat Protoc. 2014, 9(5):1160-9.
